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The Hidden Danger of Green Smoothies: Oxalate Overload

Recent headlines describing an ominous poisoning shed light on a lesser-known health risk lurking in everyday health fads: oxalate toxicity. While the story of a jilted lover who used ethylene glycol (commonly found in antifreeze) to harm his lover may seem far-fetched, the aftermath—a cascade of oxalate crystal deposits that wreak havoc on the victim’s kidneys and liver—serves as a stark reminder. the dangers posed by oxalate-rich substances.

The underwater magic of green smoothies

Amid the fervor of health-focused trends, the ubiquitous “green smoothie” has emerged as a seemingly virtuous elixir. A quick online search brings up a plethora of recipes extolling the virtues of mixing nutrient-dense greens like spinach, kale, Swiss chard and arugula with berries or almonds for alleged health boosts. Yet, unbeknownst to many, these seemingly healthy concoctions harbor a hidden danger: oxalate overload.

The oxalate conundrum revealed

Oxalate, a compound prevalent in certain foods, fungal infections, and even human metabolism, poses a significant threat when consumed in excess. Oxalates, which are abundant in vegetables such as spinach, beets and nuts, can accumulate in the body, leading to a myriad of health problems.

Danger of oxalate overload

Excessive consumption of oxalates has been linked to kidney stones, a painful condition that affects millions of people worldwide. While calcium oxalate stones are a well-known consequence, oxalate’s insidious effects extend beyond the urinary tract. Oxalate crystals can infiltrate various tissues, causing inflammation, oxidative damage, and even compromising bone health and immune function.

Revealing the culprits: Foods high in oxalate

Many seemingly harmless foods are high in oxalates, including leafy greens, berries, nuts, tea and chocolate. While these staples may seem harmless, their cumulative amount of oxalates can spell trouble for susceptible individuals.

         Mitigating oxalate overload entails a multifaceted approach:
  • Risk Reduction: Strategies for Oxalate Control
  • Alleviating oxalate overload requires a multi-pronged approach:
  • Addressing fungal overgrowth: Antifungal treatment can help fight fungal infections that are involved in oxalate production, especially in conditions like autism.
  • Supplemental Support: Calcium citrate and magnesium citrate supplements help reduce oxalate absorption, while chondroitin sulfate helps prevent calcium oxalate crystal formation.
  • Vitamin B6 supplementation: Vitamin B6 acts as a cofactor in oxalate degradation, thereby limiting oxalate production.
  • Adopting a low-oxalate diet: Limiting high-oxalate foods moderates oxalate intake and offers relief from symptoms associated with oxalate toxicity.
  • Hydration: Adequate water intake facilitates the excretion of oxalates and reduces the burden on the kidneys.
    Toxicity assessment of oxalates: Organic acid test

    The Organic Acid Test (OAT) is proving to be a valuable tool for measuring oxalate toxicity, offering insight into genetic predispositions, nutritional imbalances, and fungal overgrowth that contribute to oxalate overload.

    Conclusion: Navigating the risks of health fads

    While the allure of green smoothies can be exciting, the hidden danger of oxalate overload underscores the importance of making informed dietary choices. By adopting a balanced approach to nutrition and heeding the warning signs of oxalate toxicity, individuals can protect their health and well-being amid an ever-evolving landscape of health trends.  

References

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http://www.vulvarpainfoundation.org/Lowoxalatetreatment.htm

Fishbein GA, Micheletti RG, Currier JS, Singer E, Fishbein MC. Atherosclerotic oxalosis in coronary arteries. Cardiovasc Pathol. (2008) ; 17(2): 117–123.

Levin RI, PW Kantoff, EA Jaffe. Uremic levels of oxalic acid suppress replication and migration of human endothelial cells. Arterioscler Thromb Vasc Biol (1990), 10:198-207

Di Pasquale G, Ribani M, Andreoli A, Angelo Zampa G, Pinelli G. Cardioembolic stroke in primary oxalosis with cardiac involvement. Stroke (1989), 20:1403-1406.

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Burns JR, Cargill JG 3rd. Kinetics of dissolution of calcium oxalate calculi with calcium-chelating irrigating solutions. J Urol. (1987) Mar;137(3):530-3.

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